Sclerotherapy-induced esophageal changes

                        The local inflammatory response may lead to esophageal luminal narrowing. If the region of injection therapy was circumferential, the resultant narrowing may also be circumferential, mimicking an annular carcinoma. When less extensive, the perivariceal inflammatory and hemorrhagic response produces mucosal or extramucosal defects that may vary in size and shape. These local defects secondary to edema usually regress in a few days.
                            In patients in whom the inflammatory response was more marked, or deeper in extent, esophageal strictures may develop over time. Therefore, patients with deep ulcers or sinus tracts early on in their course are more prone to develop a stricture. Most strictures are short and may be at the site of a previous ulceration. Contour defects n oted in the early post-procedure period may persist as nodules or even plaques on later follow-up examinations. Stiffening of the injected portions of the esophageal wall may also result. Dysmotility in the area, perhaps secondary to neuromuscular damage, has been reported as well.


“radiology images of Postsclerotherapy esophagogram reveals” multiple ulcerations in the proximal to middle thirds, and a long stricture distally.

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